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These data propose that the detrimental outcome observed on PFS and OS associated with circulating VEGFA levels in patients with advanced biliary tract cancer treated with cisplatin and gemcitabine may be attenuated by cediranib.
Together these data propose that an increase in COX-2 expression in ventral CA1 following trauma is likely due to its attenuated degradation.
Altogether, these data propose that two ways of activation exist in the GABAB receptor.
These data propose that one circuit for FOXP2-dependent speech and language might be not only the prefronto-caudate-loop but also the inferior fronto (Broca's area)- rubro-olivo-lateral cerebellar loop.
While the extent of oncogenic dependency to a single copy loss of miR-212 and subsequently increased HB-EGF in JHU022 requires further investigation, these data propose a novel mechanism that abnormal regulation of EGFR ligands by genetic gain or loss of miR-containing loci may promote carcinogenesis.
Taken together these data propose key functions for Brk in breast tumour development and progression.
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The present work builds on these data, proposes additional modifications and attempts to automate it, in order to meet the requirements and capacities of modern autoanalyzers.
In their discussion, Ahmed et al. (1994) present an alternative interpretation of these data, proposing that many synapses originally assigned to inhibitory targets are potentially on excitatory cells.
These preliminary data, proposes microRNAs as potential biomarkers for migraine with applications in patients' stratification, diagnosis or perhaps therapeutic aspects of migraine.
These early data proposed that the major structural protein is RebB and that RebA may act as a scaffold to facilitate the polymerization process whereas RebC may act as a transcriptional regulator (Heruth et al. 1994).
These results support data proposing a role for Fabp7 in sensorimotor gating function and emotional control (40).
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com