Exact(4)
Hepatitis B virus (HBV) DNA levels are used to predict the response to therapy, determine therapy initiation, monitor resistance to therapy, and establish treatment success.
It is important to define the contribution of CSCs to relapse on endocrine therapy, determine their sensitivity to existing agents or identify the unique signalling pathways that sustain their clonogenic potential.
The objective of this study was to determine the virologic response to ART in a cohort of HIV-infected Ugandan infants (children less than 12 months of age) as measured by HIV viral suppression after 6 months of therapy; determine the level of immunologic recovery as measured by increase in CD4 cell counts; and determine the proportion of infants developing ART-related adverse events.
Increasing efforts to understand genetic events should allow us to perform the following functions: Classify breast tumours according to the signalling pathways that are disrupted and to predict prognosis and response to therapy; Determine the relevance of somatic events to prognosis and response to therapy; Generate new, targeted therapies based on target discovery.
Similar(56)
Interestingly, the combined HH + Cort therapy determined the lowest leucocyte adhesion whereas the HH therapy alone lowered the leucocyte transmigration.
Among men with early prostate cancer, the natural history without initial therapy determines the potential for survival benefit following radical local treatment.
Specifically, research published in the journal Sexual and Relationship Therapy determined that the Magic Wand operated on its low setting at a frequency of 89 Hz and at 101 Hz on its high setting.
Surgery was followed by adjuvant therapy determined by multidisciplinary team discussion with patient choice informed by appropriate guidelines (SIGN, 2007).
Initial antibiotic (meropenem with vancomycin), anti-inflammatory (corticosteroids) and supportive therapy determined a good clinical and paraclinical evolution.
Given that telavancin has the same efficacy as standard therapy, determining its place in therapy should be based on side effect profile, administration issues, and cost.
The combination of molecular-targeted therapy determined by the characteristics of individual EGFR phosphorylation events and EGFR recycling inhibition show promise in future treatments of cholangiocarcinoma.
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