Sentence examples for therapy damage from inspiring English sources

In clinical therapy, damage is commonly indirect ionizing.

But 2 years ago, a team led by cancer biologist Richard Kolesnick and radiologist Zvi Fuks of Memorial Sloan-Kettering Cancer Center in New York City showed that one of the principal side effects of radiation therapy, damage to the gastrointestinal tract, occurs because the radiation triggers apoptosis, a form of cell suicide, in the cells of the small blood vessels of the tract.

As the specific inhibitor of xanthine oxidase (allopurinol) protected against photodynamic therapy damage, it is concluded that reperfusion injury is involved in the mechanism of photodynamic therapy in the rat colon.

TEM- related poor ophthalmic treatment outcomes have been attributed to delay in uptake of eye care services while on first line TEM therapy; damage to ocular and or adnexal structures from intrinsic TEM toxicity or result of interaction with prescribed medications, and microbial contamination of TEM agent or procedure [ 2, 10- 12].

In the acute care section, we considered conservative versus operative therapy, damage control surgery was conceptualized as the first surgical intervention, multiple operations was defined as five operations or more, need of invasive ventilation, blood transfusion and massive transfusion, which was defined as ≥10 units of packed red blood cells (PRBC).

Researchers suspected that radiation therapy damages the stem cells that give rise to new neurons, which are concentrated in the hippocampus, a brain region necessary for storing memories.

For instance, a single application of X-ray therapy damages cancer cells but leaves the vasculature intact.

These findings are relevant to the mechanisms by which photodynamic therapy damages other tissues, including neoplasms, and particularly to the possible application of this treatment to brain tumours.

Therefore, another possible mechanism could be that VEGF-targeted therapies damage the vasculature, leading to enhanced tumour cell extravasation at the primary site or increased seeding at the metastatic site.

2, 3 While current therapies are able to increase OS and progression-free survival (PFS), these therapies damage normal tissue and typically fail. 1, 4, 5 As a result, there is a need for more specific and effective therapies which are able to selectively target tumor cells without damaging normal brain cells.

Finally, GITMO-WG expressed the requirement to consider only full-course therapies damaging SC mobilization, as lower doses or shorter therapies are not ascertained detrimental factors.