Sentence examples for their respective expressions from inspiring English sources
where we have replaced the constants (mathcal {A}_{n}), (mathcal {B}_{n}), and (mathcal {C}_{n}) in (25) by their respective expressions mentioned right after (20).
Where neither ( {b}_{max}=left({displaystyle {int}_0^kleft[1-{F}_{tilde{c}}(x right]dx-{displaystyle {int}_{-infty}^0{F}_{tilde{c}}(x right]dx-{displaystyle}_{intde{C}left(m,v,kright)}^{-1}(w) ) contain t and G and H encapsulate their respective expressions.
Those that were related to constitutive resistance were up-regulated or down-regulated in RHT compared with their respective expressions in TN1 at the same time points.
Three genes of interest have been identified with these criteria: AtPrx13 (0.64 ± 0.03, 0.67 ± 0.04 0.69 ± 0.04), AtPrx30 (0.38 ± 3, 0.32 ± 0.03, 0.50 ± 0.09) that were significantly down-regulated, and AtPrx55 (2.78 ± 0.31, 2.38 ± 0.26, 1.97 ± 0.24) that was significantly up-regulated in shp1, shp2 and shp1 shp 2 respectively when compared to their respective expression level in Col 0 background.
Migratory and classical dendritic cells (mDC and cDC, respectively) in thoracic lymph nodes were quantitated (top right), and their respective expression of costimulatory molecules (CD40, CD80, and CD86) compared.
Replacing p G f, n u and F G s, n x ∕ u by their respective expression in (15), we obtain an integral-form expression for the cdf of the intercell interference gain produced by one single interferer: F G n x = ∫ 0 ∞ Q 1 0 log 1 0 u x σ d B - σ d B 2 ξ exp - u d u. (16).
The goals of our study were thus to characterize the in vivo and in vitro expression of each gene and to elucidate the mechanisms underlying their respective expression.
The detailed list of the identified proteins with their theoretical molecular weights, pIs, number of matched peptides and their respective expression profiles are reported in Table S1.
Of particular interest is the close clustering of hESC-ECs and HUVECs, indicating a high degree of similarity between their respective expression profiles.
In order to further evaluate the status of NTS and NTSR1 in breast cancer we studied their respective expression in 106 IDCs.
In contrast, unsupervised classification approaches, achieve clustering of genes based on their respective expression profiles but are not intended to filter out uninformative genes.
