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The transcript ratio is shown as mean ± SEM (n = 6 experimental repeats).
This ratio is termed the transcript ratio (see Materials and Methods for details).
ABL kinase mutational analysis was carried out when the transcript ratio increased over two sequential samples or on clinical demand.
There was also no significant correlation when differences in absolute length were plotted against the transcript ratio (data not shown).
These histograms have 10 increments ranging from 0.1 to 1 on the x-axis; these increments reflect bins for the transcript ratio with the predominant isoform in the denominator.
The transcript ratio of the UTR isoforms of NOD2 upon stimulation was further analyzed by cloning and counting of PCR products (> 100 individual clones) as previously described [ 16, 17].
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Given the literature supports a role for TFs and promoters in determining transcript stability, another explanation for the effect of loss of Gln3 is that transcript C is stabilised, but there is no change in transcription per se and thus no change in the transcript ratios.
These results indicate that the microarray data reflects the transcript ratios for the majority of genes.
The transcript ratios of Pe/Pc in this group showed a homogenous distribution of increased and decreased transcript ratios.
This same small increase was also confirmed in the transcript ratios detected by the Affymetrix chips.
Moreover, the transcript ratios for different families or subunits of these proteins in whitefish and omul were not equal.
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