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The sections revealed that extracellular fibrils extended uniformly from cell surfaces in association with leaf tissue (Fig. 3A, D).
Immunohistochemistry of the sections revealed a similar proportional distribution of fiber types in subjects with the R225W mutation compared to controls (Figure 7).
None of the sections revealed neuronal loss apart from the local damage around the needle track, which was similar in toxin- and control-injected cases.
H&E staining of the sections revealed stark differences between the degradation patterns of the PBSA and the TBSA implants in vivo.
A CD31 (endothelial marker) staining of the sections revealed that the endothelial layer of leoligin- and control-treated conduits was intact.
Furthermore, accompanied by reduced vascular density, immunohistochemical staining of the sections revealed decrease of HIF-1α and VEGF protein, which was consistent with the findings in vitro.
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The sections reveal numerous features such as the sag basin, fractures, faults and folding below the basaltic top.
The sections reveal LCs to be typically rounded and well dispersed throughout the epidermis, with no noticeable overlapping of cells (Figure 5B,C).
Analysis of the section revealed that the ripple patterns are similar to sine-wave structures with a period of several hundred nanometers.
Microscopic examination of the section revealed fibrotic changes of the lung interstitium, proliferation of lymphocytes and plasma cells, with no malignant changes.
More exactly, double immunofluorescence staining of the section revealed the codistribution of significant signals of MPO and citrullinated histone in the fibrinoid necrosis of the cellular crescent in the lesion (Supplementary Figure S1).
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