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In the case of a low degree of PLAD-driven complex formation, however, cross-linking of dimeric antibodies would have an almost obligate impact on the secondary interaction of receptor2 antibody complexes and thus on receptor2 antibody chain/cluster formation.
B button is the secondary interaction button for breaking down doors or windows instead of opening it.
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This series enabled us to probe the secondary interactions that can play a role in the affinity of low-molecular-mass displacers for different stationary phases.
Because the folding properties of RNA and DNA are not exactly the same, the assembly of RNA was generally developed under a slightly different perspective due to the secondary interactions in an RNA strand.
The focus of this paper is on the study of the secondary interactions between the impurities (RNA, gDNA, proteins, LPS) in a pDNA-containing lysate and 3-amino PB controlled pore glass (CPG) matrices.
The elution profiles had an unfavorable shape at low TFA concentrations consisting of a spike in their front and a long tailing rear due to the secondary interactions for the peptide ion having very low saturation capacity.
The primary coordination sphere of germanium is still pyramidal for [GeI3]−, but the secondary interactions to neighbouring iodides result in an overall six-coordinate distorted octahedron at germanium.
Considerations that ought to be taken into account include the following: the delivery to the affected cells of corticosteroids, the uptake of the hormone by the cells, the metabolism of the hormone by the cells, the intracellular actions of the hormone, the possible secondary interactions between cells, and multihormonal actions on the final target cell.
There is, however, initial evidence that different types of TNFRSF receptor-associated signaling pathways differ in the need for secondary interaction of two or more TNFSF ligand3 TNFRSF receptor3 complexes for activation.
The need for secondary interaction of initially formed trimeric ligand receptor complexes for full TNFRSF receptor activation is nicely reflected by the ability of some per se non-agonistic TNFRSF receptor-specific antibodies to synergistically stimulate receptor signaling in concert with soluble TNFSF ligands.
This pan-specificity of inhibition is believed to derive from formation of a heterotetrameric complex with target proteases involving three types of interface: the dimerization interface, a primary substrate-like interaction, and a smaller secondary interaction between the partner ecotin subunit and the protease.
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