In the present study we used the same set of chromosome 5 sub-NILs as Johnson et al. (2012), focusing on mapping loci associated with horticultural traits and determining linkage relationships among these loci and the late blight resistance QTL.
These fully wild-type females share the same set of paternal chromosomes (derived from the hemiclone male) and a random set of maternal chromosomes and mitochondria.
This is most striking when the F1 male and female inherit the same set of P chromosomes, where 1∶1 segregating markers can only arise as a result of recombination in the P-male.
In the present study, we used the same set of sub-NILs for chromosome 5 as Johnson et al. (2012) and mapped horticultural trait QTL linked to P. infestans resistance QTL.
In the present study, we used the same set of sub-NILs for chromosome 11 as Johnson et al. (2012) and mapped QTL for multiple horticultural traits that were also linked to P. infestans resistance QTL.
It is believed that no chromosomes share the same set of IGHV gene segments in the human population.
Assuming genomes consist of a single linear chromosome, share the same set of genes, and contain no duplicated genes, we can represent them as permutations of integers where each integer corresponds to a gene.
When the male-determining gene first arose, some 320 million years ago, the X and Y were both full-length chromosomes, each bearing the same set of 1,000 or so genes.
The study of rearrangements led to the definition of common intervals (Bergeron et al., 2002; Jahn, 2011), conserved regions of a chromosome within which the same set of genes can be observed, albeit not necessarily in the same order.
The same set of strains was used to evaluate a B. anthracis chromosome-specific PCR assay that exploited polymorphisms found in the rpoB gene [ 31].
