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The first and most critical step in the rearrangement process is the induction of two DNA double-strand breaks (DSB).
This indicates that U is mainly controlled by the rearrangement process of the chain near the end surface of the shish.
The rearrangement process seems to be more favorable than the decomposition of CHA to carbene formation.
The rearrangement process generates a distribution P 0 on unmutated ancestors.
Indeed, functional clusters in mammals are significantly enriched by SINE elements as they contribute to the rearrangement process (Zhao et al. 2004).
A side effect of the rearrangement process is the large increase in the size of the inverted repetition, which may increase transposition ability by stabilizing the synaptic complex.
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Haas and colleagues (2009) also suggested that the high rate of transposon activity must have occurred more recently [ 13], supporting our hypothesis that the WGD event has preceded the rearrangement processes.
Within the scope of our current knowledge, this rearrangement process represents the most parsimonious and reasonable hypothesis.
It is hypothesized that error(s) in this rearrangement process result in the inability to mate, followed by loss of the micronucleus.
We consider that this mechanism may account for the gene duplication that occurred during the gene rearrangement process in the evolution of the ancestral mitogenome to the S. orientalis mitogenome.
Most likely, the high repeat content in these genomes have played an important role in this rearrangement process.
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