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In both cases, the models explain why the effect of IPI on threshold may be nonsignificant and at best small.
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The model explains why such styrene systems give large, monodisperse particles, whereas such vinyl acetate systems give much smaller particles.
Based on this argument, the model explains why people perform worse in a spatial relation problem with ambiguous solutions than in a problem with a single solution.
The model explains why MCF7 sub-clones transfected with HER2 or EGFR show three GFR-distribution patterns, and why the bimodal distribution pattern can be reversibly modulated by estrogen.
The model explains why sub-optimal investment in defensible space is likely and provides insights into the likely effectiveness of programs designed to encourage households to increase their defensible space.
The model explains why a community needs many cells.
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At the same time the model explains why, paradoxically, some type of T cell-B cell contact is required in the development of autoimmunity.
The model explains why individuals respond (or do not respond) to mitigate their health risks, particularly with respect to adherence to medical regimens.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com