Sentence examples for the induction of rises from inspiring English sources

From recent experiments, it may be concluded that two additional features are also shared by members of this family: the induction of rises in intracellular calcium and the involvement of cholesterol-rich membrane domains, at some (maybe several different) stages, in their toxicity mechanisms [13], [19], [30], [37] [39].

The incorporation of 2 3 mol% of metal ions in the nematic polymer matrix leads to the induction of SmA phase and rise in the clearing point.

The induction of pyrexia was confirmed by rise in temperature more than 0.5°C, while animals showed rise in temperature less than 0.5°C were excluded from experiment [ 11].

Expression of TNFR2 mRNA measured by real-time RT-PCR was increased after the induction of inflammation (Fig. 3a), rising to four times the initial level by 7 days after induction of inflammation.

Hence, upon serum stimulation of starved cells, the induction of MYC results in a rapid rise in level of Myc that dimerizes with Max and transregulate target genes.

This approach was based on the assumption that if migration of the β2 integrin molecules depends only on their calpain-mediated release from the talin cytoskeleton, then it would be expected that the induction of an artificial intracellular [Ca2+] rise by the calcium ionophore could enable the migration of integrin molecules into rafts.

Pulsed DC trackers were developed to reduce this artifact by generating pulses of DC magnetic fields, restricting the induction of eddy currents to only the rising and falling edges of the pulses [ 34].

Loss of function mutations in the Nodal gene, including deletions of regulatory elements that, lead to a reduction of Nodal levels of expression [13], reveal that the highest level of Nodal signalling is required during gastrulation for the induction of the anterior primitive streak, which gives rise to the mammalian equivalent to Spemann's organiser.

A consistent observation for the induction of this form of LTP is a rise in postsynaptic intracellular calcium ([Ca2+]i), and it is well established that the main source of the [Ca2+]i is entry through the calcium-permeable NMDA receptors (Bliss & Collinridge, 1993; Malenka & Nicoll, 1993).

The induction of mammary epithelial development during pregnancy is mediated by a rise in progesterone levels [ 3, 4].

A rapidly rising number of studies have shown that the induction of apoptotic cell death is an essential mechanism of anticancer agents activity [ 47– 49], including BE.