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The implication of these modifications for the establishment of durable resistance to inhibitors of cellulose remains to be shown.
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The implication of these things have not been fully explored.
The implication of these findings for transport policy are discussed.
The implication of these data are two-fold.
The implication of these findings is profound.
The implications of these findings for modification of dose-specific breast cancer risk by fractionation and protraction of dose are unclear, and the results could reflect effects of yet unrecognized confounding factors [ 45].
The functional implications of these modifications are not clearly defined yet; however, an increasing number of investigations suggest two possible roles.
Intense investigation into the role of post-translational modifications, specifically phosphorylation and acetylation, has now begun, driven by the potential implications of these modifications in Alzheimer's disease (AD) and other tauopathies [ 4, 11, 16, 18, 19].
Further research is required to understand the implication of Col II modification by HNE and other ECM components in OA as well as the potential of HNE-trapping drugs in OA.
We describe the implications of these regulatory systems in relation to modifications including H3K4me3, H3K79me3, and H3K36me3 that are associated with active genes and H3K27me3 and H3K9me3 that have been linked to transcriptional repression.
The implications of these findings are potentially far-reaching in that they speak to the impact that behavior modification may have on the unfavorable impact of inherited factors.
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