Typically, the hypothesis asserted varies with the spatial size, where habitat diversity is often considered the primary driver at local to landscape scales and the processes of colonization and extinction predominate at larger regional to global scales [11].
In contrast, the subfunctionalization hypothesis asserts that the functions of the ancestral gene are partitioned between the duplicated genes [43].
The practice hypothesis asserts that the primary function of play is to provide animals with the opportunity to practise and refine motor skills needed in adulthood.
The null hypothesis asserts that the genes annotated with a particular GO term are randomly distributed across the map, and this is tested empirically.
In contrast, the "duplication" hypothesis asserts that the photosystems diverged within a protocyanobacterial ancestor and subsequently underwent vertical inheritance and export, via LGT, to diverse anoxygenic photosynthetic lineages (Olson and Pierson 1987a , 1987; baymann et al. 2001; Olson 2001; Allen 2005; Mulkidjanian et al. 2006).
The developmental origins of adult disease hypothesis (often referred to as the Barker hypothesis), asserts that perturbations occurring during the perinatal period can have profound effects on the development of metabolism and other physiologic processes in adulthood [1], [2].
The second hypothesis, which we call the negative hypothesis, asserts that faculty have been diverted from their traditional role in basic research toward research with more commercial potential.
The CSC hypothesis asserts that malignancies arise in tissue stem and/or progenitor cells through the dysregulation or acquisition of self-renewal [ 44].
The first hypothesis, the reversion hypothesis (figure 1a) posits that L. a. astyanax and L. a. arthemis are sister taxa, while the second, the monophyletic mimic hypothesis, asserts that the mimetic L. a. astyanax and L. a. arizonensis form a clade (figure 1b).
