Interestingly, similar dense immune cell clusters have previously been described in fungal infections (Dubey et al., 2005), although the function of the clusters is currently unknown.
The functions of the clusters included: cell adhesion, metabolism, development, response to injury, transport, cell motility, glycolysis, cell differentiation, cell cycle, signaling, regulation of transcription, cell proliferation, proteolysis and peptidolysis (Fig. 4).
It examines the influence of individual firms' absorptive capacities on both the functioning of the intra-cluster knowledge system and its interconnection with extra-cluster knowledge.
Three members of this gene cluster have no known roles, yet the functions of the other cluster members suggest they could contribute to cancer progression.
For most, however, the function of the second cluster is still unclear, and speculation has centered on the second cluster acting as an electron donor in these cases.
Additional studies investigating the function of the second cluster along with the relevance of exogenously bound sulfur in the RimO crystal structure utilized HYSCORE and EPR of MiaB lacking the radical SAM cluster binding motif.
The importance of the filtering step can be clearly seen in Fig. 6d where the functions of the isoform clusters are much more similar to the query isoform when the filtering is applied.
In contrast to the functions of the genes in cluster 4, the genes in cluster 1 presented distinct functions such as regulation of reactive oxygen species, response to interlukin-6, and regulation of the activation of JAK2 kinase activity.
If the intercellular coupling is impaired with a low gc value, the function of the β-cell cluster is much less robust against β-cell damages.
Here, we investigated the function of the miR-183/-96/-182 cluster in breast cancer.
