Sentence examples for the elimination time from inspiring English sources

To determine the severity and/or the elimination time of OI, one should measure cholinesterase in blood, either by measuring plasma pseudocholinesterase (PCE) or by measuring the cholinesterase in erythrocytes (which is thought to reflect the cholinesterase in neurons and neuromuscular junctions).

The rapid decrease in the gadolinium concentration in the tumor, which is in the same order of magnitude than the elimination time of the chelates from the blood, is problematic to determine the most suited moment for irradiation (i.e. higher content of gadolinium in the tissue combined to the lower one in the surrounding healthy tissues).

Panobinostat is likely the most potent pan-HDAC inhibitor in clinical development and the elimination time of panobinostat is considerably longer than other HDAC inhibitor in clinical development.

However, the elimination time of SA was longer than ASA, which might explain the efficacy of AEE lasting longer than those of ASA and eugenol.

Panobinostat is likely one of the most potent pan-HDAC inhibitors in clinical development and as the elimination time of panobinostat is relatively long, prolonged histone hyper acetylation can be observed 7 d after a second dose with this compound.

A substantial reduction in the elimination time occurs for WPV3 in the scenarios we explored for Bihar (top of Table  3) but not for WUP (bottom of Table  3) even in the case of SIAs expanded to all ages.

Regarding the steroid elimination time after the pour-on application, it is concluded that the CCβ or ' t = 0 concentrations' are reached within 5 days after the first application.

This scenario reduces the WPV1 elimination time in the model by almost 2 months (Table  4).

Addition of a mOPV SIA targeted at all ages leads to considerably more prevented cases and may shift the modeled elimination time of the targeted serotype by over a year (i.e., WPV1 in Bihar in the top of Table  3).

Regarding the steroid elimination time, it is clear that 5 days after repeated injection (Fig.  7b), the steroid concentrations are still above CCβ levels.

However, 5-HD pretreatment blocked the decrease in infarct size associated with DDFPe alone, supporting an IPC-like event mediated by mitoKATP as the mechanism of DDFPe cardiac protection, as illustrated in Figure 4. Our results demonstrated that DDFPe elicits a cardiac protective effect that persists well beyond the assumed elimination time of DDFPe.