In Experiment 2 the effect was replicated using a between-subjects design that ruled out the possibility that the effect was a specific consequence of the within-subjects procedures used in the first experiment, in which mice may have come to expect sucrose at different time points within the feeding session.
Finally, the effect was replicated using a different set of faces from another database that were also equated for expression intensity, providing evidence that enhanced VSTM for angry faces is not specific to the faces used, nor due to the potential for angry faces to be more intense in expression than happy or neutral faces.
The effect was replicated in conditioned media to control for cellular responses.
Thus, the principal aim of Experiment 2 was to attempt to militate against this by examining whether the effects were replicated within subjects, who were scanned twice with the same paradigm involving a semantic decision task.
The conundrum is that we use transgenic animal models based on the amyloid hypothesis to test compounds for efficacy, and subsequently we exclude the patients whose pathomechanism is closest to the model organism, in which it is most likely that the observed effect is replicated.
In a study of tacrolimus response, CYP3A5/ CYP3A4 was identified previously to be associated with circulating drug levels and this effect was replicated in the Vanderbilt DNA biobank (BioVU) (Birdwell et al. 2012).
The canary effect was replicated at other factories around the country and in some plants certain staff were paid danger money.
The conditioning effect was replicated and as in Experiment 1 and 2, the conditioning effect was largest for brands presented after the ending (now told first).
Although the mutation has been identified in only two people, the power of the research stems from the fact that the shortened sleep effect was replicated in mouse and fruit-fly studies.
As a result, both the microsaccadic rate effect and the saccadic facilitation effect were replicated.
The life-shortening effect was replicated when hUCP3 was expressed specifically in median neurosecretory cells (mNSC), which express three of the Drosophila insulin-like peptides (DILPs).
