ERK1 proteins evolve faster than ERK2 proteins, even when the study was restricted to the mammalian clade; however amino acids diverge only at position neutral for function hence we propose that the origin of this difference originates from genomic factors such as the large gene size difference, but not from functional differences.
KDO8PS, but also the differences originating from the slightly different environment that each residue senses in the four subunits of the tetramer.
The differences originate from the use of different parameter values to identify LCR.
Our hypothesis is this difference originates from the capillary pressure −saturation relationship adopted in our numerical implementation.
This result is analysed in detail and we find that the dominant effect driving this difference originates from an identifiability problem when using inlet outlet pressure measurements for joint estimation of permeability and porosity.
Where this difference originates from, is not yet determined.
The only difference originates in near-surface structures.
However, as more clinical EEG abnormalities were also seen in AUDS than in MDE patients, it seems likely that the group difference originates from excess delta activity in AUDS patients.
This difference originates from the different background media: The refractive index (n) of glass substrate and air are 1.5 and 1, respectively.
This difference originates from the difference in the approach toward handling correlograms, i.e., the CA method handles a single correction value within a group, whereas the PS method processes each correlogram.
