The acute form of this mulishness is writer's block.
The acute form is common, with hyperthermia, depression and anorexia occurring within three to four days before presentation.
Therapy in the acute form is fairly satisfactory, but the chronic form is more difficult to treat.
In the acute form, the skin lesions generally subside in 6 to 24 hours, but they may come and go and persist much longer in the chronic form.
In the acute form, symptoms include a sudden onset of breathlessness, fever, a rapid heartbeat, cough (especially in the morning), copious production of phlegm, and a general sense of feeling ill.
In addition to the ischemic and toxic causes of acute renal failure, mention must be made of fulminating varieties of acute renal illnesses that are generally mild (e.g., acute glomerulonephritis see below) and of the acute form of immunologic rejection that can destroy a kidney irrevocably within minutes of transplantation.
The acute form of the disease is typically characterized by the presence of bilateral exudative retinal detachments.
The diagnosis of intestinal ischemia is still a major challenge in sub-Saharan Africa where the acute form of intestinal ischemia caused by intestinal obstruction is most prevalent [1 4].
(Fig. 10a, b and c) is usually a mark of extensive disease and reflects a chronic process (present insidiously over months), which has been reported to have less favorable therapeutic outcomes as compared to the acute form [41, 42, 43, 44].
In 10% of individuals, the disease progresses directly from the acute form to a symptomatic clinical form of chronic Chagas disease.
