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These data indicate that Sn captures cell-free virus and increases the rate of infection suggesting that Sn may function as an enhancer of HIV infection in the periphery.
The results, therefore, indicated that SN served as a major stimulus for Phase I, although they did not exclude the possibility that SN acted in concert with other signals released by the injured cornea.
It can be seen that Sn element exists throughout the whole films.
We suggest that Sn accumulates within the Au (or Au/Ga, respectively) particle during growth.
Hence, we speculate that Sn atoms diffuse preferentially in the space between the graphite layers.
This implies that Sn doping can increase the lattice parameter of Fe-doped In2O3[19].
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A pharmacokinetics study showed that SN-38 micelle formulations had a longer circulating time in blood than irinotecan.
The results indicate that Sn-Co nanoparticles are homogeneously attached to the surface of graphene.
Observation that Sn-doping considerably improves photodegradation under visible light has become a further substantial step forward [3 6].
It was imperative that Sn-bound HIV-1NL4-3 be the only source of virus in these trans infection assays.
The data indicates that SN-38 is significantly more active against proliferating endothelial cells then non-proliferating cells.
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