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Unlike the situation in ALBP [ 85, 86, 97] it has not yet been demonstrated that the chronic population contains unique groups that respond differently to different interventions.
Various combinations of these variables result in different richness counts that respond differently to biases (see below).
Over the past 10 15 years, it has become evident that there are multiple different subtypes of breast cancer that respond differently to chemotherapeutic and targeted therapies [ 2].
If minerals are used for dating, the necessary checks on the ages are achieved by analyzing samples from more than one location and by analyzing different grain sizes or mineral types that respond differently to disturbing events.
Relating clinical and molecular differences may allow us to define more specific subtypes of patients that respond differently to therapeutic interventions.
Interestingly, expression of each OGT isoform varies in different cell types, suggesting that each isoform may have distinct functions that respond differently to cellular signaling depending on its tissue distribution.
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As a mechanism for local sensitivities to systemic levels of 20E, we hypothesized that groups of cells that responded differently to 20E manipulations would differ in expression of ecdysone receptor (EcR).
We also catalogued the genes that responded differently to Cry1F toxin exposure in the two strains.
The interaction term was used to identify the genes that responded differently to Cry1F exposure in resistant and susceptible strains.
Clustering of epigenetic promoter signatures defined classes of EWS-FLI1-regulated genes that responded differently to low-dose treatment with histone deacetylase inhibitors.
This resulted in the identification of 474 genes (198 at greater than 2 fold) that responded differently to infection depending on prior feeding.
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