We know that loci which are higher in expected heterozygosity (He) are more useful for individual identification.
We find that loci highlighted by GWAS will often be highly correlated with the causal SNP, limiting the amount of distortion observed.
Schweizer and Stein [ 32] recently reported that loci for resistance to different diseases can associate in the same locus in barley.
Our results suggest that loci with influence on udder health may also contribute to genetic variance of longevity.
This suggests that loci within the 6p23.31 region, and particularly GABBR1 and HLA-A alleles confer high disease susceptibility.
This experimental design, however, does not exclude the possibility that loci distinct from EXPB1 could account for our results.
Since the β and g statistics assume that loci evolve under the SMM, loci Vica9/152 and Vicacg8/42 were excluded from the calculations.
Dealing with microsatellite data, the selective pressure hypothesis assumes that loci that show significant temporal changes in allele frequencies are linked to regions involved in immune response.
However, the identification of distinct QTL affecting radial growth and virulence results suggest that loci contributing to variation in growth do not contribute to variation in virulence.
Data from studies of morphological and life-history traits in plants and yeast support the distinction between polymorphisms buffering genetic and environmental variation, and further suggest that loci buffering different types of environmental variation do overlap with one another.
These results suggest that reproductive behaviour is shaped by opposing selection on males and females, and that loci influencing attractiveness and foraging were polymorphic for alleles with sexually antagonistic expression patterns prior to ML selection.
