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In addition, our findings confirm previously developed concept according to that diffusion in ionic liquids disobeys the Stokes Einstein equation and the transport behavior in ionic liquids cannot be described in terms of "classical" model of diffusion.
Mamisch et al. reported that diffusion in repair cartilage after MACI was significantly higher than in normal cartilage.
For example, a recent study demonstrated that bi-exponential models for IVIM may be used to model the DW-MRI signal decay of tumour tissues, suggesting that most tumour tissue voxels exhibit both perfusion and diffusion and that diffusion in tumour tissues is either free or restricted [ 14].
In the 1940s, KIRKENDALL showed that diffusion in binary solid solutions cannot be described by only one diffusion coefficient.
It was found that diffusion in the anode plays a key role in the performance of a tubular SOFC.
Results in previous literature suggest that diffusion in a one-dimensional stockpile can lead to a maximum temperature rise of ≈ 80 K.
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Although the amount of intracellular diffusible mediators in close vicinity to the Kv channel cannot be precisely quantified, the overall local cellular changes could be substantial, providing that diffusion processes in the vicinity of the Kv channels are limited.
This statement is equivalent to saying that diffusion of materials in metamorphism is a slow process, and various chemical units do not mix on any large scale.
A model that considers diffusion in UFG polycrystal undergoing recrystallization was developed.
This review summarizes our knowledge about the factors that control diffusion in thylakoid membranes and bridges structural membrane alterations to changes in mobility and function.
They find that rapid diffusion in a large lattice inhibits the non-life to life transition because fluctuations are smaller.
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