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We testify our findings by comparing with conventional equal load partitioning (ELP) strategy that is commonly used in the parallel processing literature.
To testify our speculation, we designed this study to explore whether 14-3-3ζ 14-3-3ζ 14-3-3ζom HCC cells to Tiss through exosomes andeliveredstigate the fromtional roles of 14-3-3ζ in TILs, in tHCChope of providing a new avenue to understand the mecellssms of dysfunctoonaliTILsf through in HCC microexosomesent.
Two numerical experiment results are listed in Section 4 to testify our theoretical analysis.
To testify our above theorem, we give the following numerical example.
In order to testify our hypothesis, we implemented both in vivo and in vitro experiments.
Further in-depth clinical investigations would need to be conducted to testify our hypothesis.
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Using a lumbar 5 (L5) spinal nerve ligation (SNL) pain model, we testified our hypothesis that SDH neurons and astrocytes reciprocally regulate each other to maintain the persistent neuropathic pain states.
The discovery that OCIAD2 has been enrolled in TGF β signal across CDH1 powerfully testified our postulation OCIAD2 could act as a downstream effector of TGF β signals.
The results above fully testified our candidate miRNAs by targeting specific genes to affect important biology process of sepsis progression and further illuminate the reliability of miRNA as sepsis biomarker.
In this study, we testified our hypothesis and elucidated that the opposite effect of canonical Wnt signaling in the fully cultured medium and osteogenic differentiation medium was mainly caused by different expression level of miR-17.
Besides individual proteins such as insulin and ubiquitin, we also testified our methodology for probing protein/substrate interaction, and the calmodulin melittin complex was chosen as a test sample.
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CEO of Professional Science Editing for Scientists @ prosciediting.com