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The hypothesis tested by this study was that sheep with divergent estimated breeding values (EBV) for fleece weight differ in gut metabolism and anatomy; regardless of the level of intake.
The hypotheses tested by this study were therefore twofold: first, that common genetic variants in VEGF were associated with the occurrence of non-syndromic, non-Mendelian CVM (in particular, TOF); and second, that rarer variants affecting amino-acid sequence or consensus splice sites were associated with the occurrence of non-syndromic, non-Mendelian TOF.
Furthermore, there were only a few regions that consistently produced the segregation distortion across the two crosses tested by this study and in that of Watanabe et al. (2004).
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This hypothesis was tested by two studies in this issue of Critical Care.
In full agreement with those previous studies, estragole was the predominant headspace volatile compound in all samples tested by HS SPME in this study.
When this was tested by a subsequent study, it was observed that autologous cell lines segregated separately from heterologous ones whether copy number changes were used for re-clustering or whether the expression of resident or non-resident genes was considered for re-clustering.
These regions were not among those tested by FISH in this study.
The 317 isolates tested by PCR in this study (Table 1; includes 46 H. influenzae and 13 H. haemolyticus that were also subjected to whole-genome sequencing) were assessed in duplicate, and all runs contained appropriate positive control and no-template control reactions.
Predicting soil loss outside the range of its own database without determining appropriate different values for the factors (for example the slope factor has only been experimentally determined up to 16%, and extrapolation beyond this should be tested by experimental studies).
The model was tested by simulation studies.
This was tested by studying sterility segregation of the F2 populations.
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