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The products of thermal degradation of polyisobutylene are successfully simulated according to a radical chain reaction model including diffusion-controlled termination reactions of primary and tertiary terminal macroradicals (Rp· and Rt·) and volatile small radical (S·).
The diethylamine function of chloroquine is replaced by substituted imidazole derivatives containing tertiary terminal nitrogen.
Both t-Bu and i-Pr are produced by the intermolecular hydrogen abstraction of primary and tertiary terminal macroradicals, and the subsequent β scission of the resulting on-chain macroradicals at the skeletal CC bond of the main chain yields TTD and TVD.
Non-volatile oligomers obtained by the thermal degradation of polyisobutylene were finely fractionated and the distribution of the functional groups of each fraction was characterized by a kinetic approach including the intermolecular hydrogen abstraction of primary and tertiary terminal macroradicals (Rp· and Rt·) and volatile small radicals (S·), followed by β scission.
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Details of mammary ductal structure primary, secondary and tertiary branching, terminal end buds, lobules and ductal ectasia are readily visualized using reflectance confocal imaging.
The formation of functional groups of non-volatile oligomers by the thermal degradation of polyisobutylene is characterized by a kinetic approach including intermolecular hydrogen abstraction of primary (p) and tertiary (t) terminal macroradicals (Rp and Rt) and volatile small radicals (S), followed by β-scission.
The compounds contain a common hydrophobic tricyclic 9H-xanthene moiety and an alkyl side chain with two amino groups, one of which is a tertiary substituted terminal amine, separated by three carbons and differing only in the chemical nature of the intermediary nitrogen atom.
Secondary, tertiary, and terminal (quaternary and senary) dendrites were counted from the PVD cell body to the posterior end separately on the dorsal side, the ventral side, or both.
After 6 weeks of treatment, we noticed that mammary glands from tamoxifen-treated rats were less developed than those of untreated control animals, having fewer tertiary ducts and terminal end buds, and they could consistently be identified from a blind data set (Fig. 4).
A novel NADNJ containing a terminal tertiary carboxamide moiety was discovered that was a potent inhibitor against BVDV.
Sometimes these conformations involve a slight alteration in the orientation of small part of the molecule e.g. a terminal tertiary butyl group, or a bridging phenyl group.
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