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Sensitized dysfunction of the limbic and mesolimbic systems could account in part for many of the cognitive, affective, and somatic symptoms in MCS.
Changes in fractionations associated with uptake and storage by these different systems could account for the differences in fractionations between A. variabilis and Azotobacter vinelandii (Table 1).
Dysregulation of the P-cadherin or E-cadherin adhesion systems could account for some of these structural abnormalities, because they are important for maintaining the boundaries as is netrin/neogenin signaling.
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The simulation results indicated that the designed system could account for an impressive 15% energy demand of the room.
Sympathetic activity and local (renal) renin-angiotensin-aldosterone system could account for the association.
We postulate that the stretch-induced up-regulation of the natriuretic peptide system could account for these findings.
Many factors other than the biology of the system could account, at least in part, for the numeric differences observed.
One could propose that the feedforward three-layer structure of the nervous system could account for this observation (see over-represented triplets).
We believe a more nuanced scoring system could account for this bias and improve the accuracy of reconstruction algorithms using BSI-JC and K2P invariants.
This uptake and subsequent circulation throughout the lymphatic system could account for the enhanced immunogenicity of LCMPs over stabilized-lipid bilayer MPs or synthetic liposomes.
Although residual abnormalities of the deep venous system could account for the higher prevalence of a personal history of prior deep venous thrombosis or pulmonary embolism, the higher prevalence of a family history of thromboembolic events suggests that an as yet undescribed genetically determined hypercoagulable state or predisposition may be present in these patients.
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CEO of Professional Science Editing for Scientists @ prosciediting.com