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John et al. used an AcNPV expression system that expressed H5 HA to immunize human [11].
Interestingly, a cell culture system that expressed an EGFR mutant with a reduced ubiquitination level (to only 1%) still displayed normal internalization [53].
A reporter system mimicking the process of transcription/replication of influenza virus was constructed by Lutz et al.. Cells were transfected with a reporter system that expressed Firefly luciferase in a response proportional to the infectivity of influenza virus.
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The systems of human activity are described by Checkland as a notional system that expresses some human activity of definite purpose.
This publication describes a generic system that expresses highly soluble NA with biochemical and immunological properties close to the original virus NA.
Here, we improve the versatility of this method by developing a transgenic system that expresses Cas9 in the Drosophila germline.
By combining growth factors that enhance survival and maturation of human OLs and neurons we have generated a human co-culture system that expresses mature myelin proteins and can be further manipulated for a more complete understanding of human myelination.
We then investigated the activity of purified apo LasRLBD as a quorum-quencher of C12-HSL molecules, by employing an E. coli MT102 biosensor strain harboring the pMHLAS reporter system that expresses green fluorescent protein (GFP) upon activation of LasR by exogenous addition of C12-HSL (Supporting Information Figure S1).
To gain a quantitative understanding of the relationship between the number of DSBs and p53 activation and to investigate the cause of heterogeneity in the p53 response, we established a cellular system that expresses fluorescent reporters to quantify both DNA damage and p53 dynamics in the same living cell.
To refine our knowledge regarding the effect of Nef on dendritic cells, we developed constitutive and inducible adenovirus vector systems that express high levels of Nef in monocyte-derived dendritic cells (MDDCs).
We observed complex metabolic abnormalities in GPRC6A−/− mice involving multiple organ systems that express GPRC6A, including bone, kidney, testes, and liver.
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