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The present study was designed to determine the presence of CT-1 and the gp130 cytokine system in vascular endothelial cells and to investigate whether CT-1 stimulates synthesis and secretion of ET-1 in the vascular endothelial cells.
We study the Nox1 system in vascular smooth smucle cells and propose four potential disassembly mechanisms.
Recently, RAS is a critical system in vascular biology and recently in neuroscience.
The present study aimed to investigate the effects of the renin-angiotensin system in vascular protection mediated by PPARγ agonists.
PPARG activators may play a role in hypertension and atherosclerosis through modification of inflammation and the innate immunity system in vascular cells (13, 14).
Accumulating evidence from animal and human studies points to the involvement of the Ang/Tie system in vascular barrier dysfunction during critical illness.
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Moreover, our findings support, in a clinical setting, the proposed role of transplasma membrane electron transport systems in vascular pathobiology.
Moreover, our findings support the proposed role of TPMET systems in vascular pathobiology, which deserves further clinical investigations.
SRF3 may also have a function in the cell biology of the Golgi system and in vascular tissue pattern formation.
Importantly, endoglin has a role in the development of the cardiovascular system and in vascular remodeling [ 19] and is emerging as a marker of activated endothelial cells, and its vascular expression is limited to proliferating cells.
The aim of the study was to determine the possible role of NO-system activation in vascular and renal effects of the dopaminergic system and the probable interaction between both systems during acute volume expansion in rats.
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