Sympathetic hyperactivity may evoke a hyperdynamic and hypovolemic state [32].
Migraine is a stress-related disorder, suggesting that there may be sympathetic hyperactivity in migraine patients.
Sympathetic hyperactivity exists in both PIH and PE; however, only the latter is characterized by multiorgan involvement, particularly renal impairment.
A subset of individuals with TBI subsequently manifests paroxysmal sympathetic hyperactivity [9, 10] which presents with symptoms of tachycardia, hypertension, tachypnea, and diaphoresis.
These observations provide an evidence for the described altered balance between abnormal frontal function, cortical hypersensitivity and sympathetic hyperactivity in children with migraine.
Pryzbylkowski et al. reported the anesthetic management of two patients and demonstrated that propofol, isoflurane, desflurane, hydromorphone, and fentanyl were well tolerated, and no paroxysmal sympathetic hyperactivity was seen intraoperatively in these cases [12].
Additional immunotherapy with gamma globulin from day 13 after admission for 3 days was ineffective, and the patient presented the clinical manifestations of paroxysmal sympathetic hyperactivity, including increased heart rate and temperature from day 18.
The symptoms of AWS usually emerge 24 96 h after cessation of alcohol consumption and are characterized by sympathetic hyperactivity and metabolic and psychiatric disorders (e.g. agitation, hallucination and seizures).
Esmolol was infused for sympathetic hyperactivity.
The authors reported changes consistent with nocturnal sympathetic hyperactivity.
Paroxysmal sympathetic hyperactivity (PSH) is a syndrome of paroxysmal, episodic sympathetic hyperactivity after acquired brain injury that has been recognised for almost 60 years [ 30].
