This pattern was consistent with a pro-survival host response through ERK1/2, although it did not exclude the role of the ERK signaling cascade in promoting RVFV replication, as it has been shown for influenza virus [44].
In each of these host-parasitoid interactions, three outcomes have been observed in this system: parasitoid survival and host death, host survival and parasitoid death ( = host resistance), or host and parasitoid death.
One of these FID mutants, FID19, had a transposon insertion in Rv2456c and is important for survival in host cells.
Our results showed that silencing TsNd expression in T. spiralis inhibited significantly the larval invasion and survival in host.
Stable and soluble variants were identified by the survival of host cells on solid medium containing high concentrations of the antibiotic.
Providing the proper environment for stem cell survival and host tissue integration is crucial in realizing the full potential of these cells in CNS repair strategies.
The pcaA mutant was also compromised in long-term survival in host mice.
The contribution of the pgm locus to Y. pestis survival in host cells is well documented [56].
Similar reports of c-di-GMP mediated expression of virulence genes and survival in host have been published for Salmonella typhimurium and Pseudomonas aeruginosa [21], [22].
