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But the evidence supporting this connection appears scant.
Studies supporting this connection were published and the pulsatile characteristics of some headaches and the introduction of β-blockers in migraine prophylactic treatment may further have supported this view [2, 3].
Further supporting this connection, biosynthesis mutants with decreased IAA are mildly ethylene-resistant (Stepanova et al. 2005; Stepanova et al. 2008).
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Other epidemiology studies in Europe have supported this connection, not just with pets, but with livestock as well.
Anecdotal evidence supports this connection.
We discuss data to support this connection and make appropriate recommendations.
While independent data from further families are required to confirm the link between this phenotype and PPP1R15B variants, the molecular data strongly support this connection.
Our previous analysis of alternative splicing combined with our present examination of gene duplication in the same teleost species appears to support this connection on a genome scale.
The observation that overexpression of PARP in D. melanogaster disrupts the organization of cytoskeletal F-actin resulting in aberrant cell and tissue morphology [65] may support this connection.
Other authors have supported this connection based on the distributions of related species/population pairs of fish, as in Poeciliopsis [ 10], Notropis [ 46], Chirostoma [ 17], Ictalurus [ 19], Yuriria [ 47] and almost three events of Goodeines exchange [ 6, 21].
Some of the strongest evidence to support this connection comes from a recent study of human campylobacteriosis in Australia, where despite regular clinical use of FQs and normal rates of Campylobacter infection, there are no confirmed cases of domestically acquired FQ-resistant campylobacteriosis (Unicomb et al. 2003).
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