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To support this result we separated these histological subtypes confirming that oligodendrogliomas have more benign courses [ 8, 53, 54].

To support this result, we performed stochastic simulations with the retained chromatin-dynamics parameters to generate fluorescence distributions that we compared with the experimental flow-cytometry distributions.

To support this result, we used LAMA-84 cells transduced with vector coding for shBCR-ABL and GFP or control vector (GFP alone).

To further support this result, we monitored the mitochondrial content by time-lapse confocal imaging upon TNF- α stimulation using the specific dye, MitoTracker Green.

To try to further support this result, we transiently transfected T-FGM AhR+/+ and AhR−/− cells with wild type Cav-1-GFP and with a Cav-1-YF-GFP non-phosphorylable mutant.

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Supporting this result, we observed no requirement of Ego1 to sequester Gln3 in the cytoplasm of cells growing logarithmically in YNB-glutamine medium.

Supporting this result, we observed that addition of the chemokine CCL3, known to act on immature DCs (Sallusto et al, 1998), significantly increased both their speed fluctuations (Fig 1E and Δ V/ V0 > 0.3 in 72% of control versus 85% of CCL3-treated DCs) and mean Ca2+ activity (Fig 1F).

In support of this result, we also observed different wetability of mycelia in strains expressing constitutively activated GNA1 or deleted for gna1.

To provide further support for this result, we obtained a mutant allele of bcc-1, tm3821, in which a 517 bp region comprising exon 6 is deleted.

In support of this result, we purified capsule from WT Caulobacter, Δ CCNA_00163 ('heavy' and 'rough'), Δ CCNA_00166 (Δ hvyA, 'light' and 'smooth'), and Δ hvyA Δ CCNA_00163 ('heavy' and 'rough', see below) cells.

To further support the significance of this result, we have repeated the same analysis comparing artificial short reads of length 24 amino-acids to the full Uniprot proteins from which they were drawn (Additional file 1, Fig. S5).

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