Sentence examples for support for alterations from inspiring English sources

Studies of facial expression in other clinical groups [schizophrenia and post traumatic stress disorder (PTSD)] of people who are in remission [ 14, 15] or who are at risk of mental illness [ 16] provide tentative support for alterations in emotion expressivity serving as a trait vulnerability factor for these disorders.

Nevertheless, available studies using biopsy specimens from patients with Barrett's esophagus support a carcinogenetic role for alterations in the same pathways that caused transformation of our non-neoplastic Barrett's epithelial cells in vitro, i.e. the p16/Rb and p53 checkpoint arrest pathways, the mitogenic Ras signaling pathway, and the telomerase-dependent replicative senescence pathway.

Our data do not support a direct role for alterations in the tcdC gene as a predictor of hyperproduction of Toxin A and B in NAP1-related strains.

Although at the present time it is unclear whether the altered gene expression resulted in permanent alterations, the gene targets and molecular pathways identified at 72 hpf support a molecular basis for alterations of neurite growth and synapse formation and function that have been observed in previous studies.

In this scope, the myofiber itself constitutes one of the main niche components and our model in which it is the only mutated component strongly supports a major role for alterations coming from the myofibers in the age-related process.

This would support a role for alteration in the energy balance of the cell in induction of apoptosis with glucose deprivation or 2DG treatment.

Thus, our data do not support a role for alteration in the initiation of Ca2+ signalling with diabetes mellitus but rather point to changes in the expression or regulation of L-type Ca2+ channels, perhaps as a consequence of glycosylation of residues.

Instead, the present data may provide further support for the hypothesis that alterations in fear and aggression after OFC lesions may be related to an inability to generate outcome expectancies, as suggested by Izquierdo et al. (2005).

Growing evidence supports a role for alteration of synaptic function in AD.

Support for clinical targeting of these alterations will require extensive in vitro and in vivo experimentation to understand whether and how the mutation functions biologically.

Further support for the clinical implication of alterations in the IRE IRP network is provided by the expression of ferroportin – the major or only iron exporter from cells – which can be altered upon loss of its 5′ IRE, as seen in polycythaemic mice (Ref. 92).