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Some of the results obtained from this analysis support data reported by other authors in comparable series.
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The results from our study do not support the data reported by Glueck et al. [ 6] and Zalvaras et al. [ 25] concluding that statistically significant relationship between ONFH and MTHFR C677T polymorphism is related to Caucasian subjects.
Our results are similar to those published by Marcel et al. [ 25] in type 2 diabetic patients (the annual increase of PWV was 0.11 m/sec) and support the data reported by De Angelis et al. [ 40], which suggests that arterial stiffness and its progression is greater in women with type 2 diabetes mellitus.
Our hypothesis was supported by data reported by other investigators, who were able to improve tissue perfusion in septic patients [ 5] or during cardiopulmonary bypass [ 6] with intravenous administration of nitroglycerin.
These observations support the recent data reported by other investigators showing that EGFR antagonists deplete the activation of both PI3 kinase and MAP kinase in human head and neck cancer cell lines (Bancroft et al, 2002) and human breast cancer cell lines (Normanno et al, 2002).
This hypothesis clearly explains the paradoxical opinions on the size-dependent immunity and has also been supported by the data reported by several research groups.
This result is supported by the data reported by Warlters et al. showing that 0.1 and 11 μM vincristine exhibited the same level of cell toxicity in MKN45 cells [ 43].
In support of this suggestion are data reported by Calafat et al. (2008), who found measurable levels of BPA metabolites in > 90% of urine samples from a representative cohort of the U.S. population.
In support of this hypothesis, the data reported by Kopjar and colleagues suggest that pretreatment with a combination of amifostine and melatonin prevents gamma irradiation-induced DNA damage in human peripheral blood lymphocytes in vitro [ 79].
This work was supported by Qidong Cancer Registry which collects the data reported by the health workers in the Qidong Health Care System, and was partially supported by the special funds of the Ministry of Finance and the Ministry of Public Health of China: Tumor Follow-up Registration Programs (MF2008-293, 2009–193, and 2010 90).
The data reported by the Montgomery study are thus inconclusive in supporting or refuting the epidemic model.
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