Sentence examples for sunburn cell from inspiring English sources

The biological responses to UV can be immediate and transient (inflammation, sunburn cell formation, pruritus) or delayed and chronic (photo-aging, immunosuppression, carcinogenesis).

ACR, American College of Rheumatology; co-loc, co-localisation; F, female, LE, lupus erythematosus; M, male; SBC, sunburn cell; UVB, ultraviolet B light.

Some of these effects include sunburn cell formations, basal and squamous cell cancers, melanoma, cataracts, photoaging of the skin, and immune suppression.

10 In a previous study using 16 subjects, we reported that 10% glycolic acid, applied 6 days/week for 4 weeks, enhanced the sensitivity to UVB radiation, measured as increased sunburn cell formation, and also lowered the minimal erythemal dose (MED).

BSA = bovine serum albumin; DAB = diaminobenzidine; EDTA = ethylenediaminetetraacetic acid; FcγR = Fc gamma receptor; FITC = fluorescein isothiocyanate; H&E = haematoxylin eosin; HRP = horseradish peroxidase; Ig = immunoglobulin; MED = minimal erythemal dose; PBS = phosphate-buffered saline; SBC = sunburn cell; SD = standard deviation; SLE = systemic lupus erythematosus; UVB = ultraviolet B light.

Alterations that disturb the skin barrier function in either stratum corneum lipid metabolism or protein components of the corneocytes are involved in the development of various less- or more-severe skin diseases, including erythema, edema, hyperplasia, "sunburn cell" formation, skin aging, contact dermatitis, atopic dermatitis, psoriasis, and carcinogenesis.

Moreover, after a single UVB stress at 1800 mJ/cm2, we can detect in the epidermis reconstructed with the TRIM29 shRNA cell line the presence of sunburn cells (Figure 7C), probably resulting from a higher stress-sensitivity of the cells lacking TRIM29.

For instance, a single exposure of human skin grafts to 500 mJ/cm2 UVB induces sunburn cells in the suprabasal layers of the epidermal tissue, 24 h after the exposure [11].

If the keratinocyte incurs irreparable damage, apoptosis ensues, generating sunburn cells in the epidermis [ 3].

Biomarkers measured were erythema, cyclobutane pyrimidine dimers, and sunburn cells (apoptotic keratinocytes).

In sun-damaged skin, the apoptotic keratinocytes are known as sunburn cells.