Exact(3)
For example, the results of this analysis are generally consistent with the interpretation that the copper smelter strike resulted in an 8.5-month reduction in fine sulfate PM, contributing to a concurrent reduction in mortality.
In the present study, we did not consistently observe a significant association between nonspecific PM2.5 mass or sulfate PM and hospital admissions, as has been found in most previously published studies.
As reported by Trijonis and Yuan (1978) and Trijonis (1979), the 1967 1968 strike led to measurable regional reductions in suspended sulfate PM and increases in visibility throughout the Southwest states.
Similar(57)
Some of the genes implicated in adverse health effects on exposure to ozone and sulfate-associated PM are toll-like receptor 4 (Kleeberger et al. 2000), proinflamatory cytokines (Ohtsuka et al. 2000), and tumor necrosis factor-α (Yang et al. 2005).
We also employed published data on mass-weighted size distributions for specific chemical constitutes of PM, sulfate and nitrate, to predict PM I/O ratios in the central Taiwan region; the resulting values ranged from 0.22 to 0.43 and 0.27 to 0.36 for sulfate and nitrate, respectively.
There also is evidence that exposure to other sources of PM2.5 including reentrained PM, sulfate from shipping and long-range transport, and construction dust contributes to adverse health as well.
Other components of PM2.5, such as sulfates and organic PM, are more homogeneous over the study region.
Whereas most studies that examined these associations found that sulfate-rich secondary PM was more strongly associated with mortality and hospital admissions than nitrate (e.g., Ito et al. 2006; Mar et al. 2000, 2006; Sarnat et al. 2008), a few studies have reported that nitrate in PM2.5, rather than sulfate, was significant predictor of mortality (Fairley 1999; Ostro et al. 2011).
Both of these studies found associations between increased levels of exposure to ambient PM and sulfate air pollution recorded at central city monitors and annual average mortality from cardiopulmonary disease, which at the time combined cardiovascular and pulmonary disease other than lung cancer.
Nitrates and sulfates are related to PM as they contribute to secondary particles.
But sulfates— i.e., PM components from industrial emissions had comparable effect as BC on dilation of the brachial artery [ 6].
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