C-fos, as a protooncogene, has been shown to act as an inducible transcriptional factor and suggested to regulate the expression of the downstream response genes [6], [19], [20].
During the last decade, microRNAs (miRNAs), which are short (∼22 nt) noncoding RNAs have been suggested to regulate mRNA expression levels and translational efficiency and play a fundamental role in a number of human disease states, including vascular disease [4], [5].
G-box was suggested to regulate gene expression in response to phytohormones and abiotic stimuli [ 33].
AR signaling has been suggested to regulate Nkx3.1 expression (31, 33, 35, 36).
REST has been suggested to regulate the expression of lncRNAs which could be involved in neurodegeneration and cancer [ 128].
Stem cell factor and TNF- α are suggested to regulate BLT2 expression (Lundeen et al, 2006; Qiu et al, 2006).
MuRF3 was recently shown to interact also with FHL2 and suggested to regulate its expression as an E3-ubiquitin ligase (Fielitz et al, 2007).
Moreover, the Wnt pathway has also been suggested to regulate prominin-1 expression, 46 concordantly with its central regulation for LGR5 expression in intestinal crypts.
Actin dynamics have a major role in determining spine structure (Dillon and Goda, 2005; Sekino et al, 2007), and have also been suggested to regulate the functional expression of LTD and LTP (Cingolani and Goda, 2008).
Recently, the ApiAP2 transcription factor family has been suggested to regulate stage-specific gene expression in the human parasite Plasmodium falciparum [3], [4].
