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More recently, involvement of intrinsic disorder in molecular recognition has been suggested to involve at least two possible mechanisms [ 3].
It is suggested to involve the dissociative adsorption of NO and subsequent oxygen exchange with the surface in parallel to CO oxidation by the surface oxygen species.
The brine imbibed non-uniformly, and the mechanism is suggested to involve the formation of a water-wet bi-layer between the oil and the hydrophobic chalk surface.
Netrin-1 is a laminin like protein that has recently been suggested to involve in the pathophysiology of atherosclerosis and treatment targets.
Multiple activities reported for curcumin, including inhibition of the stress-induced activation of AP-1, have been suggested to involve the anti-oxidant properties of curcumin.
Some of the recognition processes are suggested to involve ancillary lipid-binding scaffolds or regulators as well as dynamic protein protein interactions determined by bilayer architecture.
The underlying mechanism has been suggested to involve a decrease in protein synthesis in order to conserve O2.
The mechanism is suggested to involve ROS and can be inhibited by the iron chelating agent deferoxamine.
Mycobacterial species are known to induce MMP-9 expression and MMP-9 induction in macrophages is suggested to involve Cyclooxygenase-2 (COX-2) dependent signaling events [9].
While dozens of diseases have been suggested to involve immune abnormalities, for most, the underlying cause of the aberrant immunoresponse remains unclear.
Persuasion is suggested to involve a special form of learning.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com