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Sugar allocation is based on both source-to-sink transport and intracellular transport between the different organelles [2, 3] and depends on actual plant requirements [4].
It thus appeared that overexpression of the ZmWRI1 increased the total sugar allocation into the embryo.
AtMYB61 regulates water and sugar allocation and is mainly expressed in sink tissues.
This is in agreement with the roles of the studied transporters in sugar allocation between source- and sink-organs.
It regulates sugar allocation between source and sink tissues in connection with plant growth and development (O'Hara et al., 2012).
They are distributed through the plant via sugar transporters, which are involved not only in sugar long-distance transport via the loading and the unloading of the conducting complex, but also in sugar allocation into source and sink cells.
Similar(53)
It is known that sugar metabolism and allocation between different physiological functions is intimately associated with flowering transition in many plant species.
The phr1 mutant shows impairment in a broad range of Pi-deficiency responses, including decreased accumulation of anthocyanin, starch and sugars, altered Pi allocation between root and shoot, and decreased response of Pi starvation-induced genes [ 25, 31].
Therefore, it is now clearly established that not only the loading and the unloading of the conducting complex, but also the allocation of sugars into source and sink cells is controlled by sugar transporters mediating the transport of sucrose [ 7- 9], reducing monosaccharides [ 10], or polyols [ 11- 13].
In stems, decreased levels of sucrose were not associated with increased levels of hexose sugars, suggesting that warming influences carbohydrate metabolism and/or allocation differently in different organs.
It is well established that such a monosaccharide transporter is involved in a variety of biological processes related to phloem function, resource allocation, plant defence and sugar signalling [ 53].
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