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When treated with low concentrations of H2O2 in the absence of reducing substrates, there was a rapid loss of up to 35% of its peroxidase activity.
When the coated surfaces were compared to uncoated substrates, there was no statistically significant difference in cell viability in the response of two different human osteoblastic cell lines — Saos-2 and MG-63.
In intact PBICs from septic patients, with endogenous substrates, there was a gradual increase in cellular respiration that was 73% higher after 1 week compared with controls (P = 0.003).
When the CDC Rib-P reference (ANA #12) sample was tested at a dilution of 1 100 by IIF on commercial HEp-2 cell substrates, there was weak cytoplasmic staining on slides from all three manufacturers.
Using specific peptide substrates, there was an increase of 54% in the proteolytic activity of caspase-1, 84% of caspase-8, 98% of caspase-3 151% to caspase-6 and 177% of caspase-9, in the gastrocnemius muscle of animals bearing the MAC16 tumour (up to 25% weight loss), in relation to muscle from animals bearing the MAC13 tumour (1 5% weight loss).
In addition, when cells were cultured on the polymer-coated substrates, there was no significant difference in the cell viability from the control, methanol-treated surface, and the commercial adhesive resin AE (Scotchbond) coated surfaces, suggesting that the polymer surface did not cause any toxic effect to the fibroblast cells.
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In all the substrates there were at least ten amino acids between the phosphorylatable tyrosine substrate and the proline rich motif, which was adequate for SH3 domain docking and phosphorylation by Hck and v-Src in a previous study [37].
Lifestyle by itself was not significant (P = 0.5346), but substrate (P < 0.0001) and the lifestyle*substrate interaction (P < 0.0001) were highly significant, indicating that on at least one substrate there was a significant difference between activities of pathogens and non-pathogens.
On the (110) substrates, there were two SiNSs that had different tilt angles with the substrate.
On these substrates, there are open faults in the wiring, which are electrical disconnections.
Besides the known substrates, there were also several novel enriched protein candidates, one of which was a metalloenzyme α-enolase (ENO-1).
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com