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Subsequently, we selected the 12 hub nodes in the top 5% with respect to degree and their first neighbour nodes, to construct a hub network (Fig. 6B) and then used CFinder to detect cliques based on the Clique Percolation Method.
Subsequently we selected 20 muscle cytoskeletal genes (according to the FLYBASE gene ontology database at http://flybase.bio.indiana.edu/) showing at least 2-fold higher expression in CF5 vs. PA14 thoracic inoculation.
Subsequently, we selected bins with signals above background.
Subsequently, we selected 30 SNPs with a predefined upper MAF cutoff.
Subsequently, we selected hmx1 and six7 for further analysis by in situ hybridisation.
Subsequently, we selected patients who had contacted their GP for diabetes mellitus (ICPC code T90) in 2001 (n = 9,313).
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Subsequently, we purposively selected participants in order to further elaborate emerging hypothesis, some of which were particular to specific denominations.
Subsequently, we randomly selected 29 individuals from the simulated population to form the metamorphic sample, and we estimated FIS for these individuals.
Subsequently, we randomly selected one sequence per species, to reduce the overrepresentation of a few species (e.g., humans, mouse, zebrafish etc).
Subsequently, we followed a selected subgroup of diabetic patients with elevated cIMT over 2 years [ 8].
Once we determined which TKIs had the most significant reversal effect, such as imatinib and nilotinib, we subsequently selected three concentrations (1, 2.5 or 5 µM) for each TKI to determine whether their reversal effects were concentration-dependent to paclitaxel, vincristine and doxorubicin.
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