Exact(1)
Separate codes for adherent placenta may be useful given increases in the frequency of caesarean delivery; such a code was added to the ICD10 - Australian modification in 2002 (O43.2* Morbidly adherent placenta including placenta accreta, increta and percreta), enabling subsequent study in that population [ 34].
Similar(59)
A subsequent study in 2004 [7], found that the evidence for a single major gene affecting the condition was not conclusive despite a heritability estimate for the phenotype of 0.73, and that this model did not completely explain the inheritance of inherited deafness in this breed.
A subsequent study in Queensland demonstrated that the timing and location of 47 melioidosis cases was generally correlated with rainfall across northern Australia, with a case cluster associated with post-cyclonic flooding.
A subsequent study in isolated mouse hearts showed that the GLP-1 receptor agonist Exendin-4 conferred cardioprotection, and that GLP-1 (9–36) conferred protection to a lesser extent.
A subsequent study in GEMM of PDA indicated that nab-paclitaxel increases the intratumour concentration of gemcitabine by inactivation of the cytidine deaminase, an enzyme involved in the catabolism of gemcitabine (Frese et al, 2012).
A subsequent study in the same population 52 found that a coding variant in TMC8, rs7208422, was associated with SCSC and betaHPV seropositivity among controls.
Subsequent studies, in particular two that followed a double-blind, sham surgery, placebo-control design, showed variable and mostly negative results.
Subsequent studies in dogs suggested that the orbital cortex plays a role in modulation of salivation in response to auditory conditioned stimulus [4] [6].
Subsequent studies in mice revealed that a major source in the vaginal mucosa for this cytokine was NK cells [ 84].
Subsequent studies in humans suggested that variants in ALOX12 but not ALOX15 accounted for approximately 3% of the variation in human bone mass [ 53, 54].
Subsequent studies in humans indicated that ATAC/lymphotactin was primarily produced in the synovium of RA patients and so, given its role as a chemoattractant, might be a key modulator for T-cell trafficking in the pathogenesis of RA [ 9].
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