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Subsequent studies have identified abnormalities in complex I activity in PD [9], [33].
Subsequent studies have identified that CART is expressed in a number of regions known to be involved in reward and reinforcement, including the ventral tegmental area (VTA), nucleus accumbens (NAC), amygdala and hypothalamus [2], [3].
Subsequent studies have identified tumor initiating cells in breast cancer, colon cancer, melanoma, and brain tumors.
Subsequent studies have identified several targets of EBV miRNAs, both host and viral.
Subsequent studies have identified a large variety of diverse genes regulated by p53.
Subsequent studies have identified this subset as patients with cancers driven by mutationally activated EGFR (Lynch et al, 2004).
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When subsequent migraine association studies have identified more loci and we start to tackle their interplay in disease susceptibility, this might provide us with new tools for disease classification.
Subsequent gene expression studies have identified prognostic transcriptomic profiles that appear to be pertinent for the prognostication of short term relapses, specifically in estrogen receptor positive breast cancer [ 12, 13].
Although C. dubliniensis was initially identified as an 'atypical' form of the more common C. albicans, subsequent phenotypic and genotypic studies have identified it as a separate strain [2].
Definitive rodent lineage-tracing studies have identified β-cell dedifferentiation and subsequent reprogramming to α-cell fate as a novel mechanism underlying β-cell failure.
These studies have identified novel cancer targets and enabled the subsequent development of targeted therapeutics.
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