Subsequent studies demonstrated that LPS-tolerant cells do not produce inflammatory cytokines but present potent phagocytic activity and retain the ability to generate ROS [9, 15, 16].
However, subsequent studies demonstrated that alveoli recruit continually throughout the entire inflation curve in both animal models [96, 97] and in humans.
In addition, their subsequent studies demonstrated that SUMO1 modification of Sp1 could recruit RNF4 as an ubiquitin E3 ligase that subjects SUMOylated Sp1 to proteasomal degradation (Wang et al., 2011).
Subsequent studies demonstrated that the mechanism of the control on different Au nanoparticle sizes as a function of the amount of citrate is intimately related to the pH values, because different pH values will determine the formation process of the Au nanoparticles [42].
Subsequent studies demonstrated that RKIP binds only negatively charged phosphatidylglycerol in liposomes and lipid monolayers [12].
Subsequent studies demonstrated that this structure had the characteristics of a polysaccharide capsule.
Subsequent studies demonstrated that it binds to and regulates dozens of other proteins unrelated to the cytoskeletal motors.
Subsequent studies demonstrated that seizure activity itself can be neurotoxic, and that the primary mechanisms involve excitatory amino acid receptor mediation of excitotoxic insults [3] [6].
Subsequent studies demonstrated that surgical removal of the thymus in neonatal mice reduced the numbers of mature T cells in the periphery as well [39].
Subsequent studies demonstrated that in addition to acylated ghrelin, des-acyl ghrelin also stimulates lipid accumulation in human visceral adipocytes [63].
However, subsequent studies demonstrated that in vitro stimulation does not faithfully reflect the ability of pDC to produce IFNα in vivo [47].
