Sentence examples for subsequent deficit from inspiring English sources

Estimation from the deficit, however, is not straightforward, because usually one can identify screened cohorts only at aggregate rather than individual level, and it takes some years after screening before the subsequent deficit becomes observable.

When K-12 is grown with valine as a nitrogen source, valine accumulation results in positive inhibition of the branched chain amino acid synthesis pathway and a subsequent deficit of isoleucine and leucine.

Observational studies in humans have found associations between overstimulation in infancy via excessive television viewing and subsequent deficits in cognition and attention.

This conclusion is supported by studies showing that bronchial hyperresponsiveness, independent of current asthma symptoms, is associated with subsequent deficits in airway function growth during childhood.

Moreover, the presence of functional polymorphisms and subsequent deficits in expression/processing in rodents and patients leads to signs of both central diabetes insipidus and emotional alterations, e.g. decreased anxiety-related behavior [13] [16].

The 4-back blocks were used intentionally to exceed subjects' working memory capacity, with the expectation of subsequent deficits in 2-back performance and associated involvement of limbic circuitry.

In rodents, postnatal focal freezing lesions lead to the formation of cortical microgyria similar to those seen in human dyslexic brains, and also cause subsequent deficits in rapid auditory processing similar to those reported in human language impaired populations.

Microglial activation was considered as a key feature in our in vivo model, likely to be responsible for subsequent myelination deficit.

That study, in addition to our literature review, suggests that isolated ligamentous injury causing subsequent neurologic deficit is uncommon and that the primary problem is inadequate imaging.

Alterations in levels of consciousness and subsequent attention deficit are frequent features of the critically ill population, largely because these patients receive drugs that cause these features.

Furthermore, HMCs showed a significant increase in MtDNA damage after 4 days of growth in hyperglycaemic conditions, suggesting MtDNA damage in HMCs could play a role in the subsequent bioenergetic deficit observed in these cells after 8 and 12 days of hyperglycaemia.