Sentence examples for subsequent defects from inspiring English sources

Due to the high complication incidence [2, 3, 12, 13, 14], such as pull-out of the hardware (reported up to 80%%), and subsequent defects of the skin and infection, nowadays alternative methods are available.

Furthermore, targeted recruitment of SHP-1 in lipid rafts results in dephosphorylation of LAT and subsequent defects in downstream events of TCR signaling in Jurkat T cells [ 74].

Analysis of PTP-PEST null mutant embryos revealed defects in the embryonic mesenchyme and subsequent defects in vascularization, liver develoPTP-PEST nullmutantnembryos1].

Although lens vesicles were formed in the triple conditional knockout, a range of subsequent defects included abnormal cell proliferation, lack of cell elongation, and increased apoptosis, consistent with a multitude of roles for FGF signaling during lens development.

Here, we show that dfp1-r35 cells are proficient for meiS phase, but show subsequent defects in the meiotic program including reduced spore viability, reduced prDSBs, delayed expression of meiotic genes, and disrupted chromosome segregation.

The ablation main effect triggers subsequent defects of inductive, morphogenic and tropic cellular interactions (Edelman, 1988), which join the composite ablation×age interaction that increasingly subtracts between age groups from the age main effect.

Crummell would certainly not disagree that hindrances to blacks led to this cultural derailment and subsequent defect (as he saw it).

This advancement flap is designed to include both digital neurovascular bundles and eliminates the need for a secondary procedure as it allows primary closure of the subsequent defect.

As these defects could reduce the thermal conductivity of graphene by an order of magnitude or more below its intrinsic value, some reaction conditions such as acid content, temperature, or time of exposure must be optimized in order to minimize overoxidation and subsequent defect formation [11].

Silencing of BIG1 expression induces a minor disruption of the Golgi complex, described as a looser Golgi, and a profound defect in the maturation of β1-integrin followed by a subsequent defect in the spreading of the cells [20].

We demonstrate that disruption of Dazl results in a post-migratory, pre-meiotic reduction in PGC number accompanied by aberrant expression of pluripotency genes and failure to erase and re-establish genomic imprints in isolated male and female PGCs, as well as subsequent defect in progression through meiosis.