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Even though we don't have a clear understanding of the extent of genetic material exchange between these subdivided populations, a detailed analysis of the potential effects of population subdivision would be essential.
Such a classification was carried out to study the population genetic structure within the subdivided populations.
The hypothesis implies that highly subdivided populations will harbor multiple families of diverse TEs and vice versa.
This suggests that genome size variation can build up early during speciation, and that some of this variation may even accumulate among subdivided populations.
The model of spatial expansion assumes that subdivided populations expand the distribution range and increase the total number of individuals [ 52, 53].
These results suggest that there is an interaction between selection and genetic drift causing divergence even at a phenotypic level in these small, subdivided populations.
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Another scenario that generates outlier loci happens during the spread of an unconditionally favorable allele in a spatially subdivided population.
Maruyama showed that the fixation probability is the same for spatially structured populations (Maruyama 1974), under the assumptions of a structurally subdivided population with symmetric migration and haploid individuals that evolve according to the Moran process in each subpopulation.
Fragmentation introduces additional problems by subdividing populations into small, isolated sub-populations.
The CD24+/CD29lo population can be subdivided into populations of luminal progenitors and differentiated luminal MECs, depending on the expression or absence of CD61 (β3 integrin), respectively [ 6].
Allozyme surveys in B. elegans have previously established that single copy nuclear markers are both variable and subdivided among populations [ 29, 30].
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