However, given the exploratory nature of the studies, we cannot present any statistically significant results.
Finally, as is typical for this kind of work and similar to other studies, we cannot speculate on whether or not the intention to respond to disaster actually reflects real-world behavior in the event of a disaster.
While the categories of barriers and drivers presented here have some consistency with data presented in other studies, we cannot know if the patterns observed in this study would be reproduced at another institution.
Despite our extensive studies, we cannot exclude the potential roles of TLRs in the regulation of IRF-1 and IRF-7 during HIV-1 infection.
Third, from these studies we cannot determine whether methamphetamine's uptake (methamphetamine is a substrate for monoamine transporters) is by specific reuptake sites or non-specific accumulation.
In these studies, we cannot rule out that a residual level of TLX1 activity after knockdown prevented a more profound growth inhibition.
Like all descriptive studies, we cannot make any definitive statement concerning which factors do or do not contribute to or cause CCD.
Additionally, as is the case for all population-based studies, we cannot exclude the influence of unknown confounders that may have contributed to increased reports of non-HIV associated causes of death or to changes over time in the frequency in which HIV/AIDS is listed on death certificates.
Since the quality of complete sequencing results is not known in advance and thus can vary across studies, we cannot stipulate that sequences under examination have always been well read and documented, so that multiple omissions of mutations, for whatever reason, should be anticipated (see e.g. [3]).
As with all observational studies, we cannot ascertain causality.
As in all observational studies, we cannot establish causal associations.
