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The trial findings and supportive evidence from mathematical modelling studies suggest, therefore, that PID development can occur during the course of infection (Herzog 2012).
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The absence of a mitotic phenotype with siRNA studies suggests, therefore, that depletion was not sufficient to generate this phenotype in normal cells.
The limited sample sizes can also be a drawback in postmortem brain tissue studies, suggesting, therefore, replication in an independent sample cohort.
Aminophylline and bamiphylline have also been found to inhibit IPC in human studies, suggesting, therefore, that adenosine receptors are involved in the cardioprotective effect (Carr et al., 1997; Claeys et al., 1996; Tomai et al., 1996).
The results of the present study suggest therefore, that information regarding risk factors and personal risk should also be targeted across SES groups.
The results of this study suggest therefore that solvent exposure is associated with an adverse effect on temporal resolution abilities in human subjects.
The results of this study suggest therefore that, using a single measure of disease activity with an agreed level defined as remission, biologic therapies result in high remission rates in PsA patients, greater than a comparator group of RA patients even when matched for baseline disease activity.
The brief durations used in the present study suggest therefore that it would be impossible to achieve a high rate of target-pair detections using a switching strategy and that accurate performance could only be achieved by dividing attention between the two locations.
Subsequent studies have suggested therefore that the primary toxic species in AD are lower-molecular weight aggregates of Aβ lacking the highly organized structure of amyloid fibrils.
Therefore, several studies suggest a platelet involvement in ALI.
Therefore, our studies suggest that a large ice nuclei concentration can produce dominant positive charge in the lower portion of the mixed-phase region by maintaining ice saturation.
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