This transfer leaves a hole in the 6p z orbital directed to the trans position so that the ligand bonds more strongly to compensate.[ 11] Taking the oxo as the reference group, the ligand that in principle can donate trans electron density most strongly, and hence compensate for the 6p hole the most, is the imido group, which is experimentally observed.
The face-tracking is responsive, and the webcam has a feature to compensate for strongly backlit rooms, which worked very well.
That raises hopes that Germany and some other countries will grow strongly enough to help compensate for the abysmal economic performance of countries like Greece and Italy.
There exists an upper bound to the functioning of the dosage compensation mechanism: in triple qrr mutant strains where qrr4 is among the deleted qrr genes, whereas the remaining qrr gene is upregulated, its promoter is not activated strongly enough to completely compensate for the lack of the other three qrr genes.
This early growth pattern is strongly influenced by a drive to compensate for the intrauterine effects on the fetus of the maternal metabolism associated with obesity and glucose intolerance [ 55].
However, DUSP2, DUSP4, DUSP9 [ 68], and DUSP10 [ 69] have all been found to be induced by glucocorticoid, and are more strongly induced in DUSP1-deficient cells to compensate for DUSP1-deficiency in the induction of anti-inflammatory response [ 70].
For example, haplotypes (0,0,0,1) and (1,1,1,0) are strongly correlated but changing the coefficient of (0,0,0,1) cannot compensate for the coefficient change of (1,1,1,0).
37– 39 Although a digital assistive device could compensate for cognitive or physical impairment, its acceptance is strongly hindered by one's desire to maintain an ideal self-image.
To compensate for the absence of the roof structure, the vehicle was strongly redesigned by introducing new structural members and reinforcing existing critical components.
Although it is possible that TLR3- and MyD88-dependent responses fully compensate for one another during NSV infection, our in vitro data strongly implicate the actions of a non-TLR-dependent pathway in the type-I IFN response.
